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søndag den 25. august 2019

Does OXA1L, MRM2 and MRRF protein activity compensate for complex V inefficiency in ME patient cells?

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ATP synthesis by Complex V is less efficient in ME/CFS cells. The other mitochondrial complexes work harder to compensate (1). Is this p...
torsdag den 22. august 2019

AMPD3 in ME

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Adenosine monophosphate deaminase (AMPD) converts AMP to inosine monophosphate (IMP). The AMP deaminase gene family: AMPD1, muscle (m) is...
søndag den 11. august 2019

GPR35, ATPase inhibitor IF1 and ATP synthase subunits

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The ME hypothesis "the metabolic trap" tell us that IDO function in immune cells may be compromised (1). IDO1 and IDO2 catalyze ...
fredag den 26. juli 2019

Complex V is down in ME - does it also explain Electromagnetic Hypersensitivity?

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Complex V Complex V (also known as ATP synthase or F0F1ATPase) is working less efficiently in cells from ME patients (1). Chemical Int...
tirsdag den 23. juli 2019

Complex V is down in ME - does it explain Chemical Intolerance?

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Complex V Complex V (also known as ATP synthase or F0F1ATPase) is working less efficiently in cells from ME patients (1). The ATPase I...
søndag den 21. juli 2019

Mitokondrie Complex V er dysreguleret hos ME patienter

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I mitokondrierne bliver vores mad omsat til energi i form af ATP. Dette sker via citronsyre cyklus og de fem mitokondrie komplekser. Basis v...
torsdag den 18. juli 2019

Betydning af acetyl-grupper for ME sygdomsmekanismen

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ME patienter har dysreguleret glykolyse. McGregor et al (1) mener, at denne dysregulering medfører: nedsat dannelse af acetyl-grupper hi...